Myeloperoxidase gene-463G > a polymorphism and premature coronary artery disease

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Myeloperoxidase gene-463G > A polymorphism and premature coronary artery disease

We investigated the association between myeloperoxidase gene -463G > A polymorphism and premature coronary artery disease (CAD) in two Chinese population samples: 229 patients and 230 controls. Genotypes were determined by ligase detection reaction-polymerase chain reaction sequencing and the grouping technique. We found lower frequencies of both the A/A genotype and the A allele in patients (p...

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PRISMA-combined Myeloperoxidase -463G/A gene polymorphism and coronary artery disease

BACKGROUND Myeloperoxidase (MPO) -463G/A gene polymorphism may be associated with an increased risk of developing coronary artery disease (CAD). Studies on the subject, however, do not provide a clear consensus. This meta-analysis was performed to explore the relationship between MPO gene -463G/A polymorphism and CAD risk. METHODS This meta-analysis combines data from 4744 subjects from 9 ind...

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Myeloperoxidase polymorphism and coronary artery disease risk

The myeloperoxidase (MPO) gene 463G/A and 129G/A polymorphisms have been reported to be associated with coronary artery disease (CAD), but the results remain inconclusive. This meta-analysis was designed to clarify these controversies.PubMed, EMBASE, and the Cochrane Library were used to retrieve the relevant literature up to March 2015 according to keywords. A total of 8 case-control studies, ...

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c1019t polymorphism in the connexin 37 gene and myocardial infarction risk in premature coronary artery disease

background: the c1019t polymorphism of the connexin-37 (gja4) gene is a single-nucleotide polymorphisms involved in atherosclerotic plaque rupture and atherosclerosis predisposition. we examined the association between the c1019t polymorphism of the gja4 gene and the occurrence of myocardial infarction (mi) in patients with premature coronary artery disease (cad). methods: our study recruited 1...

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ژورنال

عنوان ژورنال: Genetics and Molecular Biology

سال: 2009

ISSN: 1678-4685,1415-4757

DOI: 10.1590/s1415-47572009005000035